2 Ocak 2015 Cuma

ADOLESCENCE Conception of adolescence,

Conception of adolescence,
 
Histotrical precursors;
·      ‘to grow up’
·      The Greek Philosopher Aristotle, passionate, carried away by their impulses.
·      The first great theories of adolescence starts with Jean-Jacques Rousseau.
·      Rousseau suggested 3 features of adolescence that continue to play a prominent role in current discussions of this period, adolescence is a period heightened instability and emotional conflict that is brought on by biological maturation, biological and social changes are accompanied by a fundamental change in psychological processes, adolescence recapitulates, that is, repeats in condensed form-the earlier stages of lifee through which the child has passed.
·      When developmental psychologist began to turn their attention to adolescence at the end of the 19th century, many picked up and modified Rousseau’ ideas.
·      Among all was G.Stanley Hall, the first president of APA, Heightened emotionality and stress, deep depressions, rebirth after childhood, adolescence is more flexible than any other period of development.
 
Modern biosocial approaches to adolescence;
·      Hall’s description of adolescence as a period of storm and stress, as reflected in adolescents’ conflicts with their parents, their wildy flunctuating moods, and their risky behavior, is widely accepted.
·      However, continuing dispute  over the dgree to which adolescence is characterized by stress and conflict, there is no unified theory of adolescence!
·      Arnold Gesell, higher human traits, such as abstract thinking, imagination, and self-control make their appereance late in the development of the individual because they were acquired late in the history of the species, environment nay exert a more powerful influence during adolescence.
·      Sigmund Freud, a distinctive stage of development adolescence is the genital stage, because this is the period during which sexual intercourse becomes a major motive of behavior, emotional strominess associated with adolescence results from a psychological struggle.
·      Sigmund Freud, struggle among the 3 parts of the personality, id, ego, superego, sexual excitation that accompanies puberty reawakens primitive instincts, increases the power of the id, and upsets the psychological balance achieved during middle childhood.
·      Sigmund Freud, this ambalance produces psychological conflict, the main development task of adolescence is therefore to reestablish the balance of psychological forces by reintegrating them in a new and more mature.
·      Sigmund Freud, was alos influenced by the idea of recapitulation, he argued, when sexual maturation reawakens the oedipal urges that were repressed at the start of the middle childhood, the young person must rework this conflict under the new conditions of social life that attend sexual maturity.
 
Physical development (brain );
·      13-15 years, largely related tp parts of brain that control spatial perceptions and motor functions.
·      15+ years, changes in prefrontal cortex responsible for executive processing.
·      Reasearch has found that the limbic system, an emotional and impulsive part of the brain, develops prior to the prefrontal cortex, the rational, logical part of the brain
·      Tis in part explains some of the impulsive, emotional reactions of teens.
 
Physical development ( muscular system );
·      Muscle fibers become thicker and denser.
·      By age 17, boys can be as much as 3 times stronger than girls.
·      Men – 40% of total body mass is muscle.
·      Women – 24% of total mass is muscle.
·      Largely a result of hormonal differences.
 
Puberty;
·      The series of biological developments that transforms individuals from a state of physical immaturity to one which they are biologically mature and capable of sexual reproduction.
·      Changes needed for reproductive maturity.
·      The sequence of physical change is universal, but the timing varies.
·      Age of onset ranges between 8-15 years.
·      Puberty begins with a chemical signla from hypothalamus, they activates the pituitary gland.
·      Pituitary increases production of growth hormones, which in turn stimulate the growth of all body tissue.
·      Pituitary also releases hormones related to two gonadotrophic hormones.
·      Gonads: primary sex organs, the ovaries in females, testes in males.
·      In females, gonadotrophic hormones stimulate the ovaries to manufacture estrogen.
·      In males, gonadotrophic hormones stimulate the testes and adrenal glands to manufacture the hormone testosterone, which brings the manufacture of sperm.
·      Estrogen and testosterone are present in both sexes.
·      During puberty, both sexes experience an increase in hormones.
 
The growth spurt;
·      The first visible signs of puberty is a spurt in the rate of physical growth.
·      Although growth continues throughout puberty, adolescents reach 98 percent of their adult height.
·      The rate of growth during adolescence varies for different parts of the body.
·      As a rule, leg length reaches its peak first.
·      Males and females acquire distinctive features that characterize the two sexes.
 
Physical changes;
·      Primary sex characteristics, the parts of the body directly involved in reproduction, growth of testes and penis, growth of ovaries, uterus, and vagina.
·      Secondary sex characteristics, not necessary for reproduction, anotomical signs distinguishing males and females, breast development, changing voice pitch, beard growth, body hair.
 
Sexual development in girls;
·      Menarche, first menstruation.
·      Occurs 2 years after beginning of other visible signs, avarage age – between 12 and 13, irregular menstrual cycles first few years.
·      Ovum may not be produced during every cycle.
·      Secular trend, lower,ng of the age of menarche by 4 months per decade, due mainly to changes in diet and lifestyle.
·      Pregnancy can ocur after menarche.
·      Sperm production begins between 12 and 14
·      First ejaculation about age 13 or 14.
·      Genital development and pubic hair development precedes the end of the growth spurt.
·      Development of beard and voices changes ocur near the end of the sequence.
 
Consequences of early and late maturation;
·      Early or late maturation might have an effect on young people’s peer relations, personality and social adjustment.
·      In general, studies Show that early maturing boys seem to have a more favorable attitude toward their bodies, largely because their greater size and strength maket hem more capable athletes which brings social recognition.
·      However, not all the effects of early maturation are positive for boys.
·      Adolescents boys who reach pubety at a relatively early age also more likely to smoke, drink and use drugs.
·      The Picture also complicated for girls. But overall effects are generally negative for girls.
·      Girls who develop before their pees might be embarrassed about the change in their body.
·      Later on, are also more likely to be dissatisfied with their bodies.
·      This is because, early maturing adolescents tend to weigh more and to be slightly shorter than the maturing children when they finish puberty.
·      For some girls, early maturation brings greater social prestige based on sexual attractiveness.
·      This increased sociaal prestige may carry potential risks.
·      Late maturation may also be difficcult, especially for boys in a school in which athletics is valued.
·      Late maturing girls may feel anxiety about when puberty will hit.
·      The effects of early or later onset of puberty are mediated by the social context in which maturation takes place and by the ways others respond to the physical changes young people are experiencing.

ADOLESCENCE Depression and suicide,

Depression and suicide,


What is depression;
·      Major depressive disorder, depressed mood/irritability and/or anhedonia, reduced interest or pleasure in all or most activities.
·      Sleep or appetite disturbance.
·      Recurrent thought of death and suicide.
·      Decreased energy/psychomotor agitation.
·      Difficulties concentrating/making decisions.
·      Hopelessness.
·      Feelings of worthless or guilty.
·      Symptoms which ocur together, persist for at least two weeks anda re associated with a significant loss of ability to function.

Adolescent depression;
·      Extreme sensitivity to rejection of failure.
·      Low self-esteem and feelings of guilt.
·      Frequent complaints of physical illness such as headaches and stomahaches.
·      Frequent absences from school or poor performance in school.
·      Threats or attempts to run away from home.
·      Sad, blue, irritable and/or complaints that nothing is fun anymore.
·      Socially withdrawn
·      Can be suicidal.

Scope of the problem;
·      Mean lenght of episodes: 7 to 9 months.
·      Associated with significant, comorbidity, functional impairment, risk for suicide, substance use.

Prevalence of depression;
·      Children: prevalence range form 1.5 to 2.5%.
·      Lifetime prevalence ( up to age 18 ) 15%-20%.
·      65% of adolescents report some depressive symptoms.

Increasing prevalence;
·      Adults:15-20% rates, 2:1 female to male.
·      Age 11: incidence low, males > females.
·      Age 13: incidence rising, maes = females.
·      Age 15, 18, 21: incidence rising, males < females.

Gender differences;
·      Females tend to ruminate in their depressed mood.
·      Experience more weight-related concerns than do males.
·      Hormonal changes alter vulnerability to depression.

Influences on the development of depression;
·      Genetic influences, greater heritability if the onset of depression occured before 20 years of age, biochemistry, neuroendocrine system.
·      Social-Psychological influences, negative attributional style/cognitive style, parential depression, peer relations.

Social-psychological influences;
·      Negative attributional style and cognitive distortions, an explanatory style in which one blames oeself for negative events and views the causes of events as stable over time and as generalizable across situations.

Parential depression;
·      Heredity.
·      Parental depression may result in poor parenting.
·      Modeling certain ways of thinking and cognitive styles/maladaptive ways of thinking.

Life events associated with depression;
·      Changes in family structure.
·      Changes in school.
·      Poor peer relationships, peer rejection, bullying.
·      Death, illness, accident, or trauma.

Suicide;
·      Most people who are depressed do not commit suicide.
·      But depression increases the risk for suicide or suicide attempts.
·      Suicidal behavior in adloescents is commonly precipitated by events or challenges that the teenager finds too difficult to tolerate.
·      Even things that seem minor to an adult can be major to a young person, who does not have the life experience to put them into perspective or the coping skills that an adult has honed.
·      Problem-solving and self-regulatory skills are not yet well developed.
·      Ability to cope with stressful circumstances develops.
·      Undesirable situations can and often do change!

Facts about suicide;
·      Teen with depression are at particularly high risk for suicide and suicide attempts.
·      While teen girls attempt suicide almost twice as often as teen boys, boysa re more likely to succeed because girls usually use less lethal means and survive the attempt.
·      Approximately one-third of teen who die by suicide have made a previous suicide attempt.
·      Males use more violent means, guns, hanging.
·      Only 33 to 50% were identified by their doctors as having mental illness at the time of their death and only 15 percent of suicide victims were in treatment at the time of their death.
·      Spring and falla re the months of highest risk.
·      A estimated 80% of all those who commit suicide give some warnings of their intentions or mentions their eelings to a friend or family member.

Suicide risk factor;
·      Multiple risk factors.
·      Mental disorders-particularly mood or eating disorders.
·      Feeling of hopelessness.
·      Low self-esteem, self-blame.
·      Substance abuse disorders.
·      Family history of suicide, death a family member or close friend.
·      Impulsive and /or aggressive tendencies.
·      Divorced parents or poor family communication.
·      A history of being exposed to family violence or abuse.
·      Social isolation/alienation, including because of being gay or being bullied.
·      Loss of romantic relationship or goog friendship.
·      Unwanted pregnancy.
·      Poor grades.

1 Ocak 2015 Perşembe

ADOLESCENCE Substance use and eating disorders,

Substance use and eating disorders,
 


Adolescent substance use;
·      Critical time for onset.
·      Experimentation is prevalent; most do not develop suds.
·      İlicit drugs
·      Licit ( legal substance ): nicotine, alcohol.
·      Nearly, 63% of youngsters have tried cigarettes by 12th grade.
·      Discriminating between normal patterns of experimentation and a pattern of abuse.

What constitutes misuse or abuse?
·      Negative life consequences
·      Difficulties with school
·      Peers
·      Family
·      Physical harm

Proctative factors;
·      Positive temperament/self-acceptance
·      Intellectual ability/academic performance
·      Supportive family/home environment
·      Caring relationship with at least one adult
·      External support system that encourages prosocial values.
·      Avoidance of deliquent peer friendships.

Role of genetic heritage;
·      Heritability estimates range from 40%-60% varies with different drugs.
·      No single gene, or even a handful of genes.
·      Complex interaction between genes, especially those that influence temperament and environmental factors.
·      Current model is interactive, ‘nature via nurture’.

Environmental factors;
·      Early physical or sexual abuse.
·      Early conduct problems and agression.
·      Stress.
·      Deviant peers.
·      Drug availability/school and neighborhood.

How parents influence?
·      Parent drug use ( social learning/modeling).
·      Parent attitudes/ harsh parenting.
·      Low parental monitoring.
·      Parent connectedness.
·      Parent-chid conflict.
·      Family stress events/poor, single or teenage mother.

Psychological factors;
·      Stress and coping model
·      Youngster facing greater negative life events and perceived stress are more likely to use alcohol and other substances.
·      Substance use serve a a coping function fort he adolescent.
·      Whereas some people employ a variety of adaptive coping others rely more on the use of avoidant coping mechanisms such as distraction, social withdrawl, use of alcohol and other substances to deal with stress.

Vulnerability;
·      No single factor or theory can explain
·      Biological, psychological and social interact.

Eating disorders;
·      Anorexia nervosa
·      Bulimia nervosa
·      Binge eating disorder

Anorexia nervosa;
·      Having an intense fear of gaining weight, the fear does not decrease with weight loss.
·      Weighting less than 85 percent of what is considered normal for age and height.
·      Having a distorted image of body shape, even when extremely thin, anorexics see themselves as too fat, denial os seriousness of low body weight.
·      Typically begins in the early to middle teenage years, ofen following an episode of dieting and some type of life stress
·      Ten times more in females than males.

Bulimia nervosa;
·      Bulimics cannot control their eating by retricting like anorexics.
·      The individual consistently follows a binge and purge eating pattern, self-induced vomiting.
·      Or, laxative use, over exercising to prevent weight gain.
·      A normal weight range, a characteristic that makes bulimia more difficult to detect.

Binge eating disorder;
·      Involves frequent binge eating without purging.
·      Because they do not purge, individuals with BED are frequently overweight.
·      They feel like they lost control over how much they eat and cannot stop eating.
·      Altough BED is more common among overweight or obese individuals, it can also ocur in individuals who are normal weight.

Etilogical factors;
·      Body image, body dissatisfaction and distorted body image, weight-related teasing, pressure to be thin.
·      Cultural influences/beauty standards/social influences on young women that place too great emphasis on physical appearence.
·      Role models, idealized female images increase adolescent girls’ dissatisfaction with their bodies.
·      The role of a history of being overweight in the development of eating disorders remains unclear.
·      Parenting/family influences, negative parent-adolescent relationship, parental attitudes and beliefs concerning eating, weight and body shape, criticism of the adolescent’s weight, insecure attachment, high parental expectations, controlling and interdependent relationships.
·      Controlling and interdependent relationships; an attempt by the child to express an individual identity.
·      Psychological factors, evaluating self-worth in terms of body shape, perfectionism/high standardsi to meet these high standards they turn something that they can control: their weight.
·      Psychoanalytic theory, onset of puberty, expectations of greater autonomy and increased responsibility are implicated during adolescence, dieting prevents the appearence of a mature body, avoiding pscyhosexual maturity.